Turinabol (Chlorodehydromethyltestosterone) 10 mg – 100 Tablets
Description
Biochem Turinabol (Chlorodehydromethyltestosterone) 10 mg – Oral Anabolic-Androgenic Steroid Tablets
Biochem Turinabol 10 mg – 100 Tablets is a product containing chlorodehydromethyltestosterone (CDMT), also known as dehydrochloromethyltestosterone, an orally active synthetic anabolic-androgenic steroid (AAS) derived from testosterone.
Turinabol is commonly associated with the historical pharmaceutical name Oral Turinabol. The compound was developed during the 1960s and has been associated with historical medical applications as well as documented misuse in organized athletic doping programs.
Chlorodehydromethyltestosterone is structurally related to methandrostenolone (Dianabol). Its chemical structure incorporates a chlorine substitution at carbon 4, a double bond between carbon atoms 1 and 2, and a 17-alpha-methyl group.
These structural modifications distinguish Turinabol from testosterone and methandrostenolone and influence its oral activity, metabolism, androgen receptor interactions, and estrogenic characteristics.
Turinabol belongs to the class of 17-alpha-alkylated anabolic-androgenic steroids. The 17-alpha-methyl modification contributes to oral activity by reducing metabolic inactivation during first-pass hepatic processing. However, this structural feature is also associated with potentially serious liver toxicity.
Unlike testosterone and methandrostenolone, chlorodehydromethyltestosterone is generally considered a nonaromatizing anabolic steroid because its 4-chloro substitution inhibits conversion into estrogenic metabolites through aromatase.
The absence of aromatization does not eliminate endocrine risks. Turinabol can still influence endogenous testosterone production, reproductive function, lipid metabolism, and cardiovascular health.
Chlorodehydromethyltestosterone acts primarily through intracellular androgen receptors. Receptor activation influences gene transcription and biological processes associated with protein metabolism, nitrogen balance, and androgen-dependent tissue responses.
Turinabol has been associated with anabolic effects in historical studies and documented nonmedical use. These observations do not establish that it is safe or appropriate for bodybuilding, athletic performance enhancement, or unsupervised administration.
Oral Turinabol is a historical pharmaceutical name associated with chlorodehydromethyltestosterone. The use of the Turinabol name in a Biochem product description does not establish that the product is an authentic historical pharmaceutical preparation or an approved generic equivalent.
The Biochem Turinabol product is described as containing 10 mg per tablet, with 100 tablets per package. This represents a total labeled active ingredient content of 1,000 mg (1 gram) per package.
The stated tablet strength is not an individual dosage recommendation. Product identity, purity, excipients, and manufacturing specifications have not been independently verified.
| Specification | Details |
|---|---|
| Product Name | Biochem Turinabol |
| Alternative Names | Turinabol / Oral Turinabol / Tbol |
| Active Ingredient | Chlorodehydromethyltestosterone |
| Alternative Chemical Name | Dehydrochloromethyltestosterone |
| Abbreviation | CDMT / DHCMT |
| Labeled Strength | 10 mg per tablet |
| Package Quantity | 100 Tablets |
| Total Labeled Active Ingredient | 1,000 mg |
| Dosage Form | Oral Tablets |
| Drug Classification | Anabolic-Androgenic Steroid |
| Molecular Classification | Synthetic Testosterone Derivative |
| Chemical Name | 4-Chloro-17β-hydroxy-17α-methylandrosta-1,4-dien-3-one |
| Molecular Formula | C₂₀H₂₇ClO₂ |
| Molecular Weight | Approximately 334.88 g/mol |
| Structural Classification | Chlorinated Testosterone-Derived Steroid |
| Structural Modifications | 4-Chloro Substitution, 1,2-Double Bond, 17α-Methylation |
| Primary Receptor | Androgen Receptor (AR) |
| Receptor Activity | Androgen Receptor Agonist |
| Primary Signaling Pathway | Androgen Receptor-Mediated Gene Transcription |
| Aromatization | Generally Considered Nonaromatizing |
| Oral Activity | Supported by 17α-Alkylation |
| Primary Metabolic Organ | Liver |
| Elimination Half-Life | Reliable Modern Human Estimates Are Limited |
| Primary Biological Effects | Anabolic and Androgenic Activity |
| Historical Medical Context | Selected Conditions Involving Tissue Metabolism |
| Administration | Oral in Historical Pharmaceutical Formulations |
| Controlled Substance Classification | Jurisdiction-Dependent |
| Competitive Sports Status | Prohibited Under WADA Rules |
| Excipients | Not Confirmed for the Biochem Product |
| Manufacturing Specifications | Not Independently Verified |
1. Activates Androgen Receptors
Chlorodehydromethyltestosterone interacts with intracellular androgen receptors expressed in skeletal muscle and other androgen-responsive tissues.
Following receptor activation, the androgen receptor complex influences transcriptional processes associated with protein metabolism, cellular regulation, and androgen-dependent physiological responses.
Its effects depend on systemic exposure, tissue characteristics, receptor expression, and individual endocrine status.
2. Influences Protein Metabolism
Anabolic-androgenic steroids can influence protein synthesis and protein turnover through androgen receptor-mediated signaling.
Chlorodehydromethyltestosterone has historically been associated with anabolic activity and changes in nitrogen balance.
These biological effects do not establish that nonmedical use is safe or that changes in body composition necessarily translate into long-term health benefits.
3. Has Anabolic Biological Activity
Turinabol is classified as an anabolic-androgenic steroid because it can influence tissue metabolism and androgen-dependent physiological processes.
Its structural modifications distinguish it from testosterone and other orally active anabolic steroids.
Descriptions of Turinabol as a compound producing exclusively anabolic effects without androgenic consequences are scientifically misleading.
4. Does Not Readily Undergo Aromatization
The chlorine substitution at carbon 4 inhibits the aromatization pathway associated with conversion of certain androgens into estrogens.
Consequently, chlorodehydromethyltestosterone is generally characterized as a nonaromatizing anabolic steroid.
This property does not establish that it prevents estrogen-related adverse effects from other medications or that it is an appropriate treatment for estrogen imbalance.
5. Retains Androgenic Biological Activity
Despite structural modifications affecting its pharmacological profile, Turinabol retains androgen receptor activity.
Potential androgenic adverse effects include acne, increased skin oiliness, scalp hair loss in susceptible individuals, and virilization in women.
Certain virilizing effects, including voice deepening, may be irreversible.
6. Can Suppress Endogenous Hormone Production
Exogenous anabolic-androgenic steroid exposure can suppress the hypothalamic-pituitary-gonadal axis.
This may reduce gonadotropin secretion and endogenous testosterone production, potentially affecting fertility, testicular function, and other endocrine processes.
The extent and duration of suppression vary and cannot be predicted solely from the labeled tablet strength.
7. Influences Hepatic and Cardiovascular Regulation
Turinabol is a 17-alpha-alkylated oral anabolic steroid, a classification associated with important hepatic safety concerns.
Potential adverse effects include cholestatic liver injury, abnormal liver function, adverse HDL and LDL cholesterol changes, hypertension, and increased cardiovascular risk.
The absence of aromatization does not make Turinabol a liver-safe or cardiovascular-safe anabolic steroid.
Chlorodehydromethyltestosterone carries significant health risks, including liver injury, cholestasis, peliosis hepatis, hepatic tumors, adverse lipid changes, cardiovascular complications, endocrine suppression, infertility, and androgenic adverse effects. Anabolic-androgenic steroids may also cause psychiatric symptoms and potentially irreversible virilization. Exposure during pregnancy can harm fetal development, and use in adolescents can interfere with normal growth and maturation.
The 10 mg tablet strength describes the labeled quantity of chlorodehydromethyltestosterone per tablet. It does not establish an appropriate dose, administration frequency, treatment duration, or safe exposure level.
Turinabol is an orally active synthetic anabolic-androgenic steroid whose principal mechanism involves androgen receptor activation and subsequent regulation of gene transcription.
Its biological activity differs from peptide hormones, growth hormone secretagogues, and GLP-1 receptor agonists because it acts through intracellular steroid hormone receptors.
Step 1 – Oral Administration and Gastrointestinal Absorption
In historical pharmaceutical formulations, chlorodehydromethyltestosterone was administered orally.
Following gastrointestinal absorption, the compound enters systemic circulation.
Its 17-alpha-methyl structural modification contributes to resistance against metabolic inactivation during first-pass hepatic processing.
Step 2 – Distribution to Androgen-Responsive Tissues
Following absorption, chlorodehydromethyltestosterone reaches tissues expressing androgen receptors.
These include skeletal muscle, reproductive tissues, skin, bone, and other androgen-responsive structures.
Distribution and biological responses depend on the compound's pharmacokinetic characteristics and individual physiology.
Step 3 – Binding to Intracellular Androgen Receptors
Chlorodehydromethyltestosterone interacts with intracellular androgen receptors, influencing receptor activation and androgen-responsive gene expression.
The resulting response depends on tissue characteristics, receptor availability, systemic exposure, and endocrine status.
Step 4 – Regulation of Gene Expression and Metabolic Activity
Activated androgen receptors influence transcriptional processes associated with protein metabolism, nitrogen balance, and androgen-dependent physiological functions.
The compound's 4-chloro substitution limits aromatization, distinguishing its estrogenic profile from that of testosterone and methandrostenolone.
Nevertheless, androgen receptor activation can produce both anabolic and androgenic effects.
Step 5 – Hepatic Metabolism and Elimination
Chlorodehydromethyltestosterone undergoes metabolic processing involving the liver.
Its metabolism produces multiple metabolites, some of which have been studied extensively in anti-doping analysis.
Reliable modern human pharmacokinetic data are limited, and commonly circulated elimination half-life estimates should not be treated as definitive.
The presence of detectable metabolites after exposure does not establish the duration of pharmacological activity or verify the characteristics of a particular Biochem-labeled preparation.
Turinabol is historically associated with oral solid-dose pharmaceutical formulations.
Their formulation characteristics may include:
- Chlorodehydromethyltestosterone as the labeled active ingredient.
- A labeled strength of 10 mg per tablet.
- A package quantity of 100 tablets.
- A total labeled active ingredient content of 1,000 mg.
- Synthetic testosterone-derived steroid structure.
- A chlorine substitution at carbon 4.
- A double bond between carbon atoms 1 and 2.
- A 17-alpha-methyl structural modification.
- Androgen receptor agonist activity.
- Anabolic and androgenic biological effects.
- Limited conversion into estrogenic metabolites.
- Oral solid-dose presentation.
- Hepatic metabolism and potential liver toxicity.
- Possible endocrine suppression and cardiovascular adverse effects.
- Excipients selected for tablet manufacturing and stability.
- Formulation-dependent dissolution and absorption.
- Packaging intended to protect tablets from environmental exposure.
- Product identity, purity, and content uniformity requiring analytical verification.
The precise formulation, excipient composition, tablet appearance, and manufacturing specifications of Biochem Turinabol 10 mg have not been confirmed.
The presence of 100 tablets does not establish an appropriate treatment duration or individual prescription. The Biochem product should not be represented as authentic historical Oral Turinabol or as an approved interchangeable generic without independent verification.
United States
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Canada
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United Kingdom
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Germany
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France
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Spain
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Italy
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Portugal
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Netherlands
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Switzerland
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Austria
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Sweden
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Norway
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Denmark
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Finland
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Poland
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Czech Republic
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Romania
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Greece
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Ireland
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Colombia
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Argentina
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México
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Perú
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Venezuela
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Ecuador
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Biochem Turinabol (Chlorodehydromethyltestosterone) 10mg | 100 Tablets | Oral Anabolic Steroid
Learn about Biochem Turinabol 10mg, a 100-tablet product labeled as containing chlorodehydromethyltestosterone, an oral anabolic-androgenic steroid associated with androgen receptor activation, protein metabolism, and significant hepatic and cardiovascular risks.
Biochem Turinabol (Clorodehidrometiltestosterona) 10mg | 100 Tabletas | Esteroide Anabólico Oral
Conoce Biochem Turinabol 10mg, una presentación de 100 tabletas etiquetada como clorodehidrometiltestosterona, un esteroide anabólico-androgénico oral relacionado con la activación del receptor de andrógenos, el metabolismo de proteínas y riesgos hepáticos y cardiovasculares importantes.
biochem-turinabol-chlorodehydromethyltestosterone-10mg-100-tablets
biochem-turinabol-clorodehidrometiltestosterona-10mg-100-tabletas
Biochem Turinabol Chlorodehydromethyltestosterone 10mg package labeled as containing 100 oral tablets of chlorodehydromethyltestosterone, a synthetic chlorinated testosterone-derived anabolic-androgenic steroid with androgen receptor activity.
Biochem Turinabol Clorodehidrometiltestosterona 10mg presentación etiquetada como 100 tabletas orales de clorodehidrometiltestosterona, un esteroide anabólico-androgénico sintético derivado de la testosterona con actividad sobre el receptor de andrógenos.